Publication Date

2014

Abstract

Rationale: The energy sensor AMP-activated protein kinases (AMPK) is thought to play an important role in regulating myocardial fatty acid oxidation (FAO) via its phosphorylation and inactivation of acetyl coenzyme A carboxylase (ACC). However, studies supporting this have not directly assessed whether the maintenance of FAO rates and subsequent cardiac function requires AMPK-dependent inhibitory phosphorylation of ACC.

School/Institute

Institute for Health and Ageing

Document Type

Journal Article

Access Rights

ERA Access

Access may be restricted.

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